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Trichomonas gallinae

articles by
Dr. Gordon Chalmers
Karl Frank



The diagram of the flagellate protozoan Trichonomas gallinae can be seen at the left with its undulating membrane and a central axostyle. There may be as many as 2 dozen strains of this organism infecting our racing pigeons with each strain being resistant or susceptible to some antibiotics. It may also be assumed that each of those strains exhibits a different level of virulence. The typical yellow growths in the mouth, pharynx, and crop as seen on the right are much less frequent than was the case in the past. Although these cheesy masses have become rare in Europe's racing pigeon scene, the infection trichomoniasis is still regarded there as the largest inhibitor to today's racing pigeon performance.
The wide spread use of antibiotics in the 1970's and 1980's has almost been successful in exterminating these strains which left an easily visible symptom of their presence. This "extermination" coincided with the emergence of various strains of this organism to which the pigeon's body does not react with a violent immune response and create these cheesy masses. Without an immune response, these more virulent strains are invisible to the naked eye but can easily invade the body, attacking and destroying the liver and various other internal organs.
Today's fancier may very well assume himself to be safe from trichomoniasis in the absence of cheesy masses in his birds or he may resort to periodic blind "cures" with antibiotics just in case... Either alternative is problematic because the bird's racing performance may be severely affected in the first case or the blind cures may lead to the development of resistant strains making the administration of the antibiotic ineffective. Only microscopic examination of the crop's contents will show the presence or absence of Trichomonas gallinae and also the relative concentration of this protozoan.
Some veterinarians, e.g. Tiberius Mohr, would prefer to rinse the crop and examine this liquid with a phase contrast microscope in looking for these flagellates or he may even culture this liquid in a special medium and thereby concentrate the specimen. The advantage of this system is that even a very low presence of trichomonads can be detected and they can also be tested for antibiotic resistance.
In the absence of such a specialized laboratory I need to be content following a much simpler method which still gives useful results. A cotton swab is fluffed up a bit and immersed in distilled water. The excess fluid is removed and the swab is inserted into the crop. All walls of the crop are contacted by using a swirling motion with the swab which is then withdrawn and the fluid held in the cotton is expressed onto a slide with the help of a spatula or bunt knife. We add a cover slip and have a look at the slide under a magnification of 300 X. These Protozoa can be seen to move about in a somewhat jerky wobbling motion.

Since it is counterproductive to assist in the creation of micro - organisms resistant to available antibiotics, it would be ideal if we could use only antibiotics to which the organism in question is sensitive to. However, antibiotic sensitivity tests regarding Trichomonas are not readily available in many locations. The solution is, at least partially, to examine crop smears microscopically not only before treatment but also a couple of days after treatment just to make sure that the antibiotic used is still effective against the micro - organism in question. A recent study done in the faculty of veterinary medicine in Barcelona found some Trichomonas organism resistant to all the nitroimidazole drugs tested. Similar results were obtained by a study done in Utrecht (Netherlands) where it was also found that the dosage commonly recommended for Ronidazole is usually too low and needs to be multiplied many times for the drug to be effective.

The route of infection is usually via contaminated drinking water which may happen in the basket toward the release point of any race. It would just take a couple of infected birds drinking from the trough and floating some of their protozoan guests into the common drinker. They can multiply relatively fast in their new host, especially since there is not much competition in a clean bird. Such infection will inevitably lead to reduced performance for which the bird is often blamed and culled. Some may look at culling for becoming infected with Trichomonas as a tragedy. Any bird can become infected, isn't that correct?
Very little is known about immunity to trichomoniasis in pigeons ( but please read the very informative article by Dr. Gordon Chalmers further down ) for which reason it may be advisable to have a look at factors affecting Trichomonas infection in humans:
3 microscopically indistinguishable separate species of Trichonomas organisms can establish themselves in humans, the harmless commensals Trichomonas tenax and Trichonomas hominis as well as Trichonomas vaginalis which can cause a low grade inflammation of the genito - urinary tract in both men and women. The commensal, Trichonomas tenax, has an incidence of 10-20% in some populations and is transmitted from mouth to mouth. Since it is a harmless organism it would not be advisable to administer antibiotics just because some keen medical doctor found this organism in a smear of saliva.
The other commensal, Trichomonas hominis, lives in our intestinal tract. It is quite common in the tropics and transmitted by the fecal-oral route.
Trichomonas vaginalis is a common parasite of both males and females. Coitus is the common mode of transmission. The organism is commonly found in females, surveys indicating from 25-50% of sexually active women being infected; only about 5% of men are infected. Many cases of trichomoniasis are totally asymptomatic! In fact, asymptomatic cases are the rule in males. The intensity of infection, the ph of vaginal and other secretions, the physiologic status of the vaginal and other genitourinary tract surfaces, and the accompanying bacterial flora are among the factors affecting pathogenicity. The organisms cannot survive at the normal vaginal acidity of ph 3.8-4.4.

Should we therefore not treat our racing pigeons against trichomoniasis at all?
This may be possible if you are lucky enough in that the only strain of Trichonomas gallinae present in your birds has a very low virulence.
However, it is not practical for many fanciers who keep their flying team(s) free of this Protozoan. They cannot afford the drastic decrease of performance (with the loss of a large percentage of their racing team) due to a rapid multiplication of Trichonomas organisms in their birds. They would rather choose the periodic attempt to keep these flagellates at a low concentration.
Nevertheless, a microscopic examination before and after such treatment appears to be the prudent thing to do. In such examinations one may often find "little critters" dashing about at high speeds in straight lines. They are much smaller than Trichomonas gallinae and are other protozoa called Hexamita or Spironucleus. They also can do much damage and some drugs such as Emtryl® used against canker may be ineffective against them.

The time may have come to look into more natural management practices of many diseases but particularly trichomoniasis. It has been shown by a number of veterinarians that, try as much as one may, it is not possible to remove the organisms causing canker entirely from the pigeon. There will always be some left after an antibiotics administration and these will start the cycle anew, multiplying when the growing conditions are favourable for them. Not only do we run the risk of this organism to develop immunity to our trichomonicidal drugs through repeated dosing but these drugs also have definite toxic effects on the pigeons we try to protect.
Ad Schaerlaekens wrote an interesting anecdote regarding this very dilemma:

A long time ago Belgian star Marc Roosens told me he had just one problem in pigeon sport: trichomoniasis. No matter how much he medicated, the birds could not get rid of it. Till he ‘saw the light’. After he had given the birds water the drinker was taken from the loft after just a few minutes twice a day and he kept on doing this for weeks and… believe it or not, since then the problem was solved. What he did was give trichomoniasis not a chance to stay alive."
Ad also relayed the following:
"In Germany scientist made tests:
Pigeons were given water in a common drinker, others were given water individually and the difference was spectacular. Birds that drank out of the same drinker nearly all had trichomoniasis but hardly any trichomoniasis was found among the birds which were given water individually."

Food for thought, isn't it?

Canker Revisited


When I read the title of the recent excellent Digest article ("Is There a New Strain of Canker?" - December 15, 1998) by Dr Kevin Zollars, my first reactions to the question posed were, "Yes, very likely, and what is more, there are likely more than one new one." I was pleased when I read the article itself because of the realistic and philosophical points Kevin raised. After it is read carefully several times, and well absorbed, this article should be placed prominently in the files of every fancier. It occurred to me then that, as a corollary to this key article, I might present some background information on strains of Trichomonas gallinae, the cause of canker, and their importance to all of us. I have drawn the information in this article from a number of important old and some fairly current scientific papers selected from my files. Incidentally, in the following material, when I refer to the canker organism, I will likely use the terms "Trichomonas gallinae, T. gallinae (the latter is simply a shortened form of the full scientific name), trichomonad, trichomonas and canker organisms" interchangeably -- all mean the same thing.
Infection by this organism was first identified in Europe in 1878 by a researcher named Rivolta. Many years later in the USA, a scientist named Robert Stabler, conducting research in Colorado, pioneered extensive work on the organism in pigeons -- in fact in 1938, he gave the organism its scientific name, Trichomonas gallinae.
In a 1948 publication on the subject, he noted that not all pigeons that harbor the organism die of the infection, or even have internal changes to indicate the presence of this organism. As well, he found that youngsters from some parents in a loft nearly always died of canker in a few days or weeks after hatching, whereas certain other parents, although infected, raised healthy youngsters indefinitely. Obviously these facts gave rise to the idea that there were strains with differing abilities to cause disease, a suggestion that had also been proposed by other scientists who had worked on canker in pigeons.
To test this idea, Dr Stabler then set up an experiment in which he used canker organisms that he arbitrarily designated as "strains" (see explanation in the next paragraph), from five different sources: Strain 1 from an infected wild youngster, Strain 2 from a healthy adult King, Strain 3 from a healthy adult Carneaux, Strain 4 from an adult racing pigeon that had a history of transmitting lethal canker to his youngsters and to at least three successive hens, and Strain 5 from the mouth of a peregrine falcon that had died with severe canker of the mouth. (Note that canker caused by T. gallinae occurs in birds of prey in which it is called "frounce". Broadly related organisms in this group also cause infections, variously, in the reproductive systems of humans, cattle, and sheep, and in the digestive tracts of domestic chickens and turkeys. I have also seen it in devastating outbreak form in small aviary finches in which the disease very much resembled that seen in the oral cavity of young pigeons.)
Dr Stabler defined "strain" as the particular canker organisms removed from the mouth of an individual bird, even though he recognized the possibility that any given bird might harbor more than one strain. The results he obtained seemed to justify the use of the organisms from a particular bird as "a strain", at least in terms of their ability to cause disease. He maintained the five individual strains mentioned previously by inoculating them by eyedropper into the mouths of clean pigeons, and took great care to be sure that the different strains weren't accidentally mixed. The clean pigeons he infected with these five strains came from his own loft of racing pigeons that he knew were free of canker-causing organisms.
In the first experiment, he used 25 of his own young birds, aged 6 weeks, 5 1/2 weeks, 5 1/2 months, 7 months, and 9 months, with five birds in each group. One bird in each age group was inoculated by mouth with Strain 1, one in each group received Strain 2, and so on. Results showed that the Strains 1, 4, and 5 caused severe signs of disease that ended in the death of all except two youngsters, a 7 and a 9-month-old bird infected with the Strain 4. These two birds had severe canker for over a week, but they recovered. Strains 2 and 3 either didn't produce signs of disease in the youngsters they infected, or the infection was very slight and lasted only 2-4 days.
In follow-up work, Dr Stabler showed that Strain 1 (which became known in trichomonad circles as his famous "Jones' Barn" strain) obtained from the wild youngster with canker, was the most deadly of the five strains, killing 12 of 13 birds inoculated with it in an average of 10.6 days. Over all, he was able to show that, of 119 pigeons infected successively with this potent strain, 114 (95.8%) died in 4 to 18 days. In later work, he showed that Strain 1 was deadly even if only one organism was placed in the mouths of susceptible pigeons. Obviously, this single organism multiplied rapidly into the thousands or more to cause serious illness.
These results showed that there was a marked difference in the ability of these five different strains of T. gallinae to cause disease in pigeons. These strains varied from those that caused little or no disease to those that caused high losses. Obviously, there were also strains that were intermediate in their ability to cause canker, since they were able to cause serious illness from which most birds eventually recovered.
In important later studies, Dr Stabler was able to show that mild strains of the canker organism were able to protect birds against more deadly strains, a finding that continues to have practical application today. To confirm these results, he first gave eight of his own trichomonas-free youngsters the relatively potent Strain 5 obtained from the peregrine falcon. All developed severe canker of the mouth, six birds recovered and two died. Fifty-four days after the initial infection with Strain 5, the six survivors were given the very deadly Strain 1. None of them developed evidence of disease during the following month. These six birds were then killed and examined at post mortem. There was evidence of scarring of the liver of three birds, findings that suggested infection from the previous dose of organisms. The other three birds were almost completely free of signs of infection. The only significant finding in these birds was the loss of the palatal fringe on the roof of the mouth. (Dr Stabler believed that, in every case examined, this change was highly characteristic of evidence that the canker organism was the cause.)
He then repeated this experiment with eight more clean youngsters that were first given the mild Strain 3 from the adult Carneaux. Only two youngsters developed a mild form of the disease. About a month later, all eight birds were given the deadly Strain 1. In the next three weeks, only two of the eight birds developed signs of canker. One had a mild form of the disease, and the other had a severe form from which it eventually recovered.
Post mortem examinations of these eight birds determined that tissues of seven birds were completely normal, and that the bird that developed severe canker had severe changes of canker in the liver. At the same time, as a control, Dr Stabler inoculated 13 youngsters from his own loft of trichomonas-free birds with deadly Strain 1; 12 of the 13 birds died. Thus, these experiments demonstrated that infection by a mild strain of T. gallinae conferred protection against a more deadly strain of the organism. However, the duration of that immunity wasn't determined at that time.
During the spring, summer and fall of 1950, there was a major outbreak of canker in mourning doves across much of the southern USA, with the greatest losses apparently in Alabama where it was estimated that deaths might well run into the thousands in that state. Dr Stabler obtained strains of trichomonads from several sources of these doves to see if the organisms from these doves could cause illness in pigeons. He inoculated 50,000-100,000 organisms from different doves, into each of five pigeons from his clean colony. For comparison, he inoculated only 3,000-10,000 organisms of his deadly Jones' Barn strain into another five clean pigeons, all of which subsequently died of canker of the liver. The most deadly of the strains from the doves came from a bird collected in Alabama, and like the Jones' barn strain, this one proved to be equally deadly, killing all but one of the pigeons inoculated with it. The other four strains obtained from the doves proved to be relatively mild when inoculated into pigeons, as most of these pigeons survived the infection.
The next question to resolve was this: would pigeons that survived the infections with mild strains obtained from doves, be able to withstand infection by the deadly Jones' barn strain? To test this idea, Dr Stabler inoculated all of these survivors with the Jones' barn strain. The result was that all birds inoculated with the Jones' barn strain survived, findings that indicated good protection following infection with strains from the doves.
    Did the fact that these birds survived mean that
  1. the strains derived from doves had killed off the deadly Jones' barn strain, or
  2. was the Jones' barn strain still present in these surviving birds, and if so, was it now altered so that it was now a mild strain, or
  3. was the Jones' barn strain as powerful as ever for clean birds, but unable to cause illness in protected birds??

To test these ideas, Dr Stabler collected canker organisms from birds that had a combination of a mild strain and the deadly Jones' barn strain, and inoculated these organisms into clean pigeons. The results were variable, as some of the newly infected clean birds had only mild changes of canker, whereas other birds either died of severe canker, or almost died. These results indicated that the deadly Jones' barn strain continued to be present, and equally important, was as potent as ever. Over all, of 13 birds infected, six died outright, one barely survived, and six had mild cases of canker. Incidentally, Dr Stabler reported that the Jones' barn strain typically caused the most severe disease in the liver of infected birds, whereas milder strains produced only oral infections.
Although the procedure isn't too practical for us as pigeon fanciers, Dr Richard Kocan working at the Patuxent Wildlife Research Center, Laurel, Maryland, found that blood plasma from pigeons infected with even a mild strain of T. gallinae could protect other pigeons infected with a deadly strain of the organism. Much more practically, Dr Kocan was also able to demonstrate that previously infected pigeons treated with the old anti-canker drug Enheptin, were free of the organism for as long as 16 months, yet remained immune to infection when they were inoculated with deadly strains. On this point, some of his other work showed that 172 of 313 wild pigeons and 54 of 66 mourning doves (all of the mourning doves captured were completely free of the canker organism) -- all trapped in his area, were resistant to the deadly Jones' barn strain. His conclusions: recovery from an infection with T. gallinae, even when the birds eventually completely eliminate the organism from their systems, results in long-term immunity to this parasite -- a fact that is of great importance to us as pigeon fanciers, one that we can use to advantage, especially in these days of apparent resistance by this organism to some of our modern, previously useful drugs.
The subject of drug resistance by the canker organism to modern drugs is also of major current importance to us. Almost 10 years ago, in 1990, Drs Lumeij and Zwijnenberg of Utrecht University, Holland, demonstrated the fact that canker organisms recovered from a large flock of pigeons in that country, were uniformly resistant to all of our commonly used modern drugs -- Emtryl, Ridzol, Spartrix and Flagyl. On the basis of that information, it seems likely that canker organisms in many other untested flocks of pigeons in Holland and indeed throughout Europe, and likely North America as well, could have been similarly resistant at that time, likely, as these researchers pointed out, because of the common practice among fanciers, of continually under- dosing birds with these drugs.
Although the subject of under-dosing, especially with Emtryl, is a pet peeve of mine, and I sound like a broken record on the matter, I think it bears repetition. In my travels, I find that the dosage of the 40% water-soluble so-called "Canadian" Emtryl, as recommended by several pigeon supply houses in North America is far below that recommended for pigeons by the producer of the drug. (At one time, this company sold small 3-gram packets of Emtryl, the exact dosage for one Imperial gallon - 4.55 liters). The fact that Emtryl is being recommended today at much lower dosages could certainly contribute to the problem of drug resistance mentioned in the previous paragraph, and may be a developing problem with major far-reaching consequences for us. I would remind fanciers that the correct dosage of Emtryl for pigeons, as recommended by the company, is 3 grams (or one level teaspoon) per imperial gallon (4.55 liters) of drinking water for 5-7 days. For the US gallon (4 liters), this is about 3/4 teaspoon per gallon for the same treatment period.
To avoid the problems of toxicity if birds drink excessive amounts of water especially during hot weather, try an Australian method that I know works well. Make up the correct dosage of Emtryl and place it in front of the birds at, say, the evening feeding for a couple of hours or so. After this time, throw out the medicated water and replace it with fresh water until the next evening. Repeat the correct dosage for a couple of hours or so each evening for a total of 5-7 days. This method insures firstly, that birds receive the correct therapeutic dose each day for the treatment period, and secondly, that problems with toxicity can be largely avoided. As Dr Zollars pointed out in his article, don't treat with Emtryl or other drugs of the same family during the pairing up period, because there is some suggestion that the drug can interfere with fertility. It is also a good idea to change drugs each time you feel birds need to be treated, say, Emtryl for one 5-7 day treatment period, and Ridzol for the next one, etc., all at the correct dosage.
Still on the subject of canker and treatments, some fanciers subscribe to the idea that if it's not broken, don't fix it. Dr Colin Walker, the Australian veterinarian who has written excellent articles for the Digest, seems to accept this idea. In one of his books, he states that drugs alone will never control a canker problem. He feels that it is important to allow developing youngsters enough exposure to the organism that they can develop natural resistance -- my idea for many years as well, based on the work of Dr Stabler.
Dr Walker expands on this idea by stating that if birds in the stock loft (and presumably their youngsters) did not develop canker the previous year, no treatment is needed this year. However, if canker did occur in stock birds and their youngsters last year, birds should be treated this year with a suitable drug prior to mating, and for two days every week after that. Further, he suggests co-ordinating these two-day treatments with the hatching period when trichomonad shedding is the highest. If the occasional youngster still develops canker, he recommends treating the parents and both youngsters in the nest with Spartrix or Flagyl for three days. (Note here that Dr Zollars has some legitimate concerns about short periods of treatment, etc. -- see pages 32-34 of his article.) Dr Walker also recommends avoiding the treatment of breeding pairs whose youngsters don't develop canker, so that there is no interference in the development of natural resistance.
Speaking of natural resistance, I recall that when I worked in New Zealand during the early 1980s, a medical doctor there raced pigeons, but apparently didn't treat his birds for any disease. Instead he preferred to rely on the development of natural resistance to any virus, bacteria or parasite his birds might encounter.
For the past several years, I haven't used preventive canker treatments on any of my old or young birds, and touch wood(!), so far there hasn't been a detectable problem. It is probable that the natural resistance developed in these birds by repeated exposure to the strains of canker organisms that very likely reside in my birds has (to date) been holding the disease at bay. Based on information from Dr David Marx, I have also been examining the mouths of my birds during the racing season for evidence of reddening and excess stringy mucus, findings that could suggest multiplication of canker organisms and increased irritation of the oral cavity during this stressful time. So far, on the basis of finding clean, pink throats, I haven't felt a need to treat preventively during the racing season, although it is possible that deeper areas such as the crop, which I didn't examine, may have been affected. I acknowledge the possibility, however, that if I had treated periodically for canker in spite of these normal findings, some racing performances might have improved. As far as canker is concerned, the idea "if it's not broken, don't fix it" seems to be working. If things change for the worst, I am ready to treat if I have to.
I hope that this look at the historical background of strains, along with the recent article by Dr Zollars, may stimulate thought on this subject among fanciers. As the risk of drug resistance by canker organisms (and other agents as well) increases steadily, I hope that fanciers may be better able to assess the facts surrounding natural immunity, and to use these facts to their advantage by recognizing the biological benefits of using any mild strains of canker organisms that reside in their birds as a major defense against deadly strains. In saying this, I also recognize the need to treat birds when or if the disease occurs. A combination of judicious treatment when necessary, plus strategies to allow for the development of natural resistance may well be the best approach. I also hope that information on the correct dosage of Emtryl -- and by extension, other drugs as well -- may help to reverse the trend of vastly underdosing our birds with these products.
This page was last up-dated on April 11, 2005

Below are your comments:
Sunday January 4, 2004
Robert Lynch
Tifton, GA, USA
Another very good article on one of the major problems in racing pigeons. I am a firm believer in building natural immunity and seldom if ever treat my youngbirds during their first 2-3 months or all birds during the winter. However, during the race season I feel it is necessary to treat for canker here in the hot, humid Southeastern US to be competative in both oldbirds and youngbirds.
Thursday April 14, 2005
faiz khalid
maroc, maroc
thank you for this information about "trichmonose"
Thursday April 14, 2005
Jose Castillo
Louisville, ky
Thanks for the information
Saturday October 1, 2005
Kevin Whittock
Huntly, Waikato. New Zealand
Thank you very much for an informative article. Thank goodness for the internet as there is a shortage of literature on racing pigeons in this country
Wednesday May 17, 2006
Pablo Lezcano
General Pico, La Pampa, Argentina
Thank you for this information. it was very rare to me to take this information...
Tuesday June 19, 2007
Emanuel Schembri
San Gwann, Malta (europe)
keep up the good site and good information.. I found it very useful.. Emanuel !!
Monday July 16, 2007
andrew pace
st`venera, malta
wonderful site and instructiv information thanks to youre teaching and knowlage.
Wednesday August 29, 2007
saul rincon
boulder city, Nevada
you have an wonderful site thankyou for the health tips my birds are now starting to have that extra energy and the offsprings are healthyer than ever wish me luck 2008.
Tuesday November 6, 2007
André de Oliveira
Vitória, ES-Brasil
thanks for the information
Tuesday November 13, 2007
Floyd Revan
Sandy Point, St.Kitts
I am happy that information on Tichomonas gallinae was posted. It is very interesting organism that is rarely heard of. The information is important and makes well for research material.
Thursday March 6, 2008
Spiros Kalamaras
Thessaloniki, Greece
Gentlemen you have amazing information not only about trichomonas gallinae but the entire site is valuable and precious even for the most experienced breeders. Congratulations and keep on going....
Monday September 8, 2008
Sunday March 15, 2009
Deniz Mobin
Dobrich, Bulgaria
Does Anybody Know Something About
Thursday March 26, 2009
j s
Tallinn, Estonia
I used successfully mild water solution (solution color similar to tea average darkness) of 'Kalii Permanganas' for cleaning-wiping 'yellow cheese-like substance' of pigeons mouth regularly several(!) times at day, using this solution with 'ordinary soft-edged stics for cleaning human ears', till yellow substance disappeared. Same time used antibiotic 'Trimerazin'. If ordinary pigeon food is not eatable for birds (because of exessive amount of 'yellow substance'), then you need to use very liquid form of food or liquid porridge. In worsest case (lot of yellow substance covering inner lower part on mouth) you need feed birds, using trachetobe (or soft(!) plastic tube appr. 3mm diameter), pushing it softly-carefully(!) inside from mouth, trying avoid touching 'yellow substance,. Using too much pressure can easily traumatize very sensitive parts(with 'yellow substance') of mouth - try to avoid occuring blood. Small blood you can stop with same solution 'Kalii permanganas' described above. Give bird water-soluble multivitamins and other natural things like Ginseng, Eleutherococcus a.s.o. This way i saved already some 20 pigeons. Actually all pigeons life can be saved, if problem is located in mouth area.
Friday July 24, 2009
Joseph pickstock
runcorn, england
Very informative article learnt a lot about canker infection and treatment in pigeons.
Saturday October 17, 2009
Joycelyn Jackson
savannah, georgia
thank you for this very important information you was willing to share pepole about this disease
Thursday February 25, 2010
saad lemjimer
kenitra, Morocco
un document et texte et aussi experiante perssonelle beaucoup nutretive. merci beucoup Mr de publier vos experiance, pour le public sur ce site qui est albeta classic.manifique je remerci tout ceux qui on aider a develloper cette passion qui est la colombophilie. merci
Sunday August 15, 2010
john west
dartford, england
lots of good tips very good
Sunday October 3, 2010
mohammad sh
Qom-Iran, I
thank you for this article.
Tuesday October 12, 2010
satyender singh
pune, Maharashtra
really useful info canker can be avoided by applying a strict routine to pigeons and not over feeding them
Friday December 3, 2010
Tom Makowecki
Edmonton, Alberta
Very informative video which you have added. Great information for anyone who wants to check their birds with a microscope. Thanks
Saturday December 4, 2010
Jeff Mawbey
Duncan, BC
Thanks for posting the video Karl, I found it very informative
Sunday February 13, 2011
pt gee
Manila, Philippines
thanks for the information! It was very helpful. I was looking how to treat our pigeon's canker and this article definitely provided me all the information that I need. A million thanks1
Wednesday June 1, 2011
Nils Reither, DVM
Thank you for this superb article. Very usefull indeed. However, a new appraoch to combat Cancker is by using a plant product called Berimax. It has been tested in 100 or more lofts the last 5 years or so, and it seems to be very effective, especially when other medication fails. This because it is a totally new and unused component isolated from certain plants. It is well tolerated, and many fanciers and veterinarians has started using this "natural remedy". To find ways to purchase it look at the web adresses, and
Wednesday June 1, 2011
Karl Frank
Edmonton, AB
I have found Ridzol to be very well tolerated and effective in removing the trichomonas organisms especially if the water is acidified.
Thursday June 2, 2011
Nils Reither DVM
Karl Yes, Ridzol (Ronidazol) is very effective in most cases, however like what Dr. Lumij and Dr. Zwijnenberg found around 1990, most strains of Tricho were resistant to Ridzol and other "-zoles" (including Emtryl), in the Netherlands, Europe at that time. The situation may be different in Canada. However, veterinarian Dr. Steve Weir in Catoosa, OK, and many fanciers and other veterinarians have also found most trichomonas resistant. Another concern is that Ronidazol /Rodzol / Emtryl / and partly Flagyl is carcinoganic (may cause cancer). So fanciers giving these medicines to their pigeons over many years, may well risk getting cancer form this. I therefor find it prudent to use other methods to control trihomoniasis. Water hygiene as described in the above article, combined with Berimax to combat infection in the pigeons, and using Citromed to kill bugs IN the drinking water, is a very good way to keep the flock free from disease. Kind regards Dr. Nils Reither, veterinarian, Norway
Thursday June 2, 2011
Karl Frank
Edmonton, AB
Some studies in mice and rats do indeed show that Flagyl can be carcinogenic in high enough doses given for prolonged periods of time but: 1.) There is no evidence that this is so in humans 2.) Flagyl is still the standard drug to be used for trichomonas vaginitis in humans: 3.) Flagyl is used very effectively in combination with other antibiotics in the treatment against periodontal disease in humans. Contact with flagyl or other imidazole drugs by the fancier during treatment of his pigeons can therefore be considered completely innocuous. Regarding resistance of the trichomonas organisms imidazole drugs: This can be a problem with any drug especially when such antibiotic has been administered inappropriately but I am happy to be able to say that I haven't come across such antibiotic resistance at least partly because: 1.) I use a trichomonicidal antibiotic only to birds that are positive to the organisms (checked with a microscope). 2.) I check any treated pigeon again after treatment to make sure that such treatment was effective. 3.) I NEVER treat "prophylactically".
Friday October 17, 2014
David Reynolds
grimmen, germany
I am interested to hear your opinion on using apple vinigar in the drinking water as a preventative measure, The artical was very interesting and informative thank you

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